Wow great result – 15% weight lost, WOW!!!! I think I| will ask for that over 50 MOT, just passed my 50th birthday.
19 Oct 2016 – HbA1c levl – IFCC standardised 39 mmol/mol
19 Oct 2016 – Serum cholesterol 6.1 mmol/L, Se non HDL cholesterol level 4.7 mmol/, Serum LDL cholesterol level 4.01 mmol/L,
Serum triglycerides 1.56 mmol/L
28 Sep 2015 – HbA1c levl – IFCC standardised 37 mmol/mol
28 Sep 2015 – Serum cholesterol 5.9 mmol/L, Serum LDL cholesterol level 4.06 mmol/, Serum triglycerides 1.1 mmol/L
3 Nov 2011 – Serum cholesterol 6.19 mmol/L, Non HDL cholesterol level 4.84 mmol/L, Serum LDL cholesterol level 3.91 mmol/L,
Serum triglycerides 2.04 mmol/.
3 Nov 2011 – HbA1c levl – IFCC standardised 40 mmol/mol
Ive only started jogging recently, the last two years started walking briskly but even that hurt my ancles and knees and kept giving up, started cleaning up my diet in the past 2 years and more the last year and want to do things seriously and follow Dr Mosleys experiments, BSD, 5:2 etc. I just checked and my BMI was 39 in 2004 and now is around 31 but needs to be in the 20’s. I was 115kg two years ago now about 103kg, ideal weight is around 73kg.
I do believe that those with fatty liver may convert MUCH fat or easier than normal people, I’m sure I saw one study where normal lipogenesis accounted for 3% of liver fat but those who had fatty liver that number rose to 20 or 30%, if that is the case that IS massive, I have to try and find it, I can’t believe it myself so I’ll have to look for it.
But …
https://www.ncbi.nlm.nih.gov/pubmed/14631324/
Enhanced lipogenesis appears as a major abnormality of hepatic fatty metabolism in subjects with NAFLD. Therapeutic measures aimed at decreasing hepatic lipogenesis would therefore be the most appropriate in order to reduce hepatic TG synthesis and content in such patients.
https://www.ncbi.nlm.nih.gov/pubmed/24316260
Increased lipogenesis in fatty liver patients
This could be the reason that I put on weight when on I go off the low carb diet and to the McDougal diet.
I think the med diet is good and allows a little olive oil or good oils, fish oil.
https://www.ncbi.nlm.nih.gov/pubmed/19370776/
An olive oil-rich diet decreases accumulation of TGs in the liver, improves postprandial TGs, glucose and glucagon-like peptide-1 responses in insulin-resistant subjects, and upregulates glucose transporter-2 expression in the liver. The principal mechanisms include: decreased nuclear factor-kappaB activation, decreased low-density lipoprotein oxidation, and improved insulin resistance by reduced production of inflammatory cytokines (tumor necrosis factor, interleukin-6) and improvement of jun N-terminal kinase-mediated phosphorylation of insulin receptor substrate-1. The beneficial effect of the Mediterranean diet is derived from monounsaturated fatty acids, mainly from olive oil. In this review, we describe the dietary sources of the monounsaturated fatty acids, the composition of olive oil, dietary fats and their relationship to insulin resistance and postprandial lipid and glucose responses in non-alcoholic steatohepatitis, clinical and experimental studies that assess the relationship between olive oil and NAFLD, and the mechanism by which olive oil ameliorates fatty liver, and we discuss future perspectives.